By William Montagna, Richard L. Dobson
Advances in Biology of pores and skin, quantity VII: Carcinogenesis covers complaints of the fifteenth Symposium at the Biology of epidermis, held on the Oregon local Primate learn heart on April 9/11, 1965, less than the auspices of the college of Oregon clinical institution.
This booklet consists of 19 chapters, and starts off with the mechanism of tissue homeostasis in grownup mammals and the kinetics of epidermal response to carcinogenic brokers and different epidermis irritants. The succeeding chapters care for the expansion selling results of tumors on tissues, the response trend differences among common and neoplastic epithelium, and a few organic implications of chemical carcinogenesis. massive chapters are dedicated to quite a few cancer causing agents, together with hydrocarbons, viruses, androgens, and estrogens. different chapters think of the physicochemical mechanisms of acceleration of epidermis carcinogenesis and experimental observations of environmental carcinogenesis. The mechanisms of epidermis melanoma induction as a result of ultraviolet radiation, in addition to arsenic precipitated tumors are tested. The concluding chapters describe a few varieties of pores and skin tumors, reminiscent of adnexal tumors and basal telephone epithelioma.
This e-book will end up beneficial to oncologists and researchers within the box of carcinogenesis.
Read or Download Carcinogenesis. Proceedings of a Symposium on the Biology of Skin Held at the University of Oregon Medical School, 1965 PDF
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Additional resources for Carcinogenesis. Proceedings of a Symposium on the Biology of Skin Held at the University of Oregon Medical School, 1965
SCOTT, E. J. VAN and EKEL, T. M. (1963). Kinetics of hyperplasia in psoriasis. Arch. Derm. 88, 373-381. SCOTT, R. B. and BELL, E. (1964). Protein synthesis during development: control through messenger RNA. Science 145, 711-713. SEKERIS, C. E. and LANG, N . (1964). Stimulation of messenger R N A synthesis in rat liver by Cortisol. Life Sei. 3, 169-174. SETÄLÄ, K. (1965). Differences in pharmacodynamic response to colchicine between benign and malignant epidermal hyperplasias. , suppl. 237,1-89.
The control of epidermal mitotic activity in the mouse. Proc. Roy. Soc. B 151, 517-536. BULLOUGH, W. S. and LAURENCE, E. B. (1960b). The control of mitotic activity in mouse skin. Dermis and hypodermis. Exp. Cell Res. 21, 394-405. BULLOUGH, W. S. and LAURENCE, E. B. (1961). Stress and adrenaline in relation to the diurnal cycle of epidermal mitotic activity in adult male mice. Proc. Roy. Soc. B 154, 540-556. BULLOUGH, W. S. and LAURENCE, E. B. (1964a). The production of epidermal cells. Symp. Zool.
This homeostatic mechanism only operates in tissues which normally show mitotic activity or which in an emergency are capable of it. The cells of non-mitotic tissues, like the dying cells of all tissues, possess a closed genome and their syntheses for tissue function are directed by long-lived mRNA. It is because of the closed genome that the alternative synthetic program leading to mitosis has become impossible. In non-mitotic tissues the control TISSUE HOMEOSTASIS IN ADULT MAMMALS 31 of tissue syntheses may be achieved merely by end-product inhibition.