Equine Wound Management by Ted S. Stashak

By Ted S. Stashak

Equine Wound administration, moment version is a complete, authoritative source for either theoretical and sensible details at the care of wounds in horses. Now hugely illustrated with full-color images all through, this long-awaited moment variation is considerably extended to incorporate new advancements and methods in wound therapeutic. Equine Wound administration is a necessary reference for veterinary scholars, veterinary surgeons, veterinary dermatologists, and equine and massive animal veterinarians.

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Extra resources for Equine Wound Management

Sample text

The autocrine nature of epithelial cell–derived IL-1 is emphasized by the fact that it additionally induces the cell to synthesize IL-1, transforming growth factor (TGF)-α and KGF. Interleukin-1 also influences matrix synthesis and remodeling via stimulation of fibroblast proliferation and enhancement of collagenase production. 31 Other ILs have also been attributed a role in wound repair. 34 Tumor Necrosis Factor-α (TNF-α) Tumor necrosis factor-α (TNF-α) is produced by a variety of cell types including macrophages, T cells, mast cells, and epithelial cells, and exerts principally inflammatory effects.

54 Keratinocyte Growth Factor (KGF) Keratinocyte growth factor-1 is a member of the rapidly growing FGF family; it is sometimes referred to as FGF-7 and resembles bFGF (FGF-2). 56 It stimulates migration and especially proliferation of these cells, but also affects differentiation of early progenitor cells within dermal appendages in the wound bed and adjacent dermis. 58 Platelet-Derived Growth Factor (PDGF) Platelet-derived growth factor (PDGF) is a family of isoforms consisting of homo- or heterodimers of products of two genes, the PDGF A-chain gene and PDGF B-chain gene.

Expression is up-regulated during early wound repair in response to tissue hypoxia, and it correlates with the density of granulation tissue developing within the wound. While VEGF activity is considered essential to optimal wound angiogenesis via stimulation of ECM degradation, and proliferation, migration, and tube formation by endothelial cells,70 it is not critical to wound healing. 72 Other Tissues Involved in Traumatic Wounds Tendons and Ligaments The reader is referred to Chapter 10 for detailed information on managing tendon lacerations.

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